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Re: How Spike Protein Causes Bizarre, Long Blood Clots 

By: Decomposed in 6TH POPE | Recommend this post (1)
Sun, 06 Nov 22 7:37 AM | 32 view(s)
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Msg. 37086 of 60008
(This msg. is a reply to 37085 by Fiz)

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fizzy:

Re: “Based on gene analysis, COVID-19 and post-mRNA vaccine injury have a common molecular mechanism. ”
I wish they would get to the point. I'm GUESSING it means that it was really stupid for Pfizer and Moderna to inject people with crap that manufactures the poisonous part of the SARS-Cov-2 virus. Do you agree with me on that?







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The above is a reply to the following message:
How Spike Protein Causes Bizarre, Long Blood Clots
By: Fiz
in 6TH POPE
Sun, 06 Nov 22 6:49 AM
Msg. 37085 of 60008

http://iowaclimate.org/2022/11/05/blood-clotting-from-spike-proteins/

The original article was behind a paywall, so I did some sleuthing to find this.

You can get the spikes from Covid, itself, or from the "vaccines". Per this article, it is the spike protein itself which causes the clots.

Excerpt:
"Spike Protein Is the Smoking Gun

There is clinical evidence that the SARS-CoV-2 spike protein has been detected in clots retrieved from COVID-19 patients with acute ischemic stroke and myocardial infarction.

Recent research conducted by cardiologists from the University of Colorado sheds light on the crucial role of spike protein in the pathology of COVID and COVID vaccine-related injuries.

They analyzed seven COVID-19 patients and six mRNA vaccinated patients with myocardial injury and found nearly identical alterations in gene profiling patterns that would predispose them to clotting state, inflammation, and myocardial dysfunction.

In other words, regardless of whether the myocarditis was caused by the virus or vaccine,
similar changes were exhibited in the expression of genes responsible for prothrombotic state
in response to spike protein, inflammation, and myocardial dysfunction.

Based on gene analysis, COVID-19 and post-mRNA vaccine injury have a common molecular mechanism. The altered genes pattern includes down-regulation in ACE2, ACE2/ACE ratio, AGTR1, and ITGA5, and up-regulation in ACE and F3 (tissue factor)."


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